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Gregory F. Grauer, DVM, MS, DACVIM

Dr. Grauer received his DVM degree from Iowa State University in 1978. He then completed his postgraduate training (internship, residency, and master’s degree) at Colorado State University between 1978 and 1982. Dr. Grauer obtained his specialty board certification in Internal Medicine in 1983. After his postgraduate training, he became a member of the faculty at the School of Veterinary Medicine, University of Wisconsin for seven years and then returned to the Department of Clinical Sciences at Colorado State University where he served as professor and section chief of Small Animal Medicine until 2000. Dr. Grauer is professor and head of the Department of Clinical Sciences at the College of Veterinary Medicine, Kansas State University. Dr. Grauer’s areas of clinical and research interest involve the small animal urinary system.

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Video: What are the most common causes of glucosuria?

Dr. Gregory Grauer discusses what causes this abnormal result, including a new culprit that pet owners may be giving as a treat.

Video: Which drugs can hurt the kidneys?

Dr. Gregory Grauer addresses which drugs commonly used in dogs and cats are potentially nephrotoxic.

Video: Controlling OA pain in dogs

Dr. Gregory Grauer weighs in on whether NSAIDs can be used in dogs with liver or kidney disease.

The nuts and bolts of proteinuria (Proceedings)

Persistent proteinuria of renal origin is an important marker of chronic kidney disease (CKD) in dogs and cats. Unfortunately, due to the high incidence of false-positive results for proteinuria on the urine dipstick screening test and proteinuria associated with lower urinary tract inflammation, positive reactions for urine protein are quite common and therefore often disregarded.

Modulating proteinuria and hypertension of chronic kidney disease (Proceedings)

By altering pre-glomerular resistance, healthy kidneys can maintain relatively stable glomerular capillary pressures despite variations in systemic blood pressure. This process is termed "renal autoregulation". Autoregulation can be reduced when renal disease results in loss of nephrons.

The nuts and bolts of azotemia (Proceedings)

Azotemia is defined as increased concentrations of urea and creatinine (and other nonproteinaceous nitrogenous substances) in the blood. The interpretation of serum urea nitrogen and creatinine concentrations as a measure of renal function requires a knowledge of the production and excretion of these substances.

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